dc.contributor.author | Sekar, Durairaj | |
dc.contributor.author | Tusubira, Deusdedit | |
dc.contributor.author | Ross, Kehinde | |
dc.date.accessioned | 2023-08-22T08:48:37Z | |
dc.date.available | 2023-08-22T08:48:37Z | |
dc.date.issued | 2022 | |
dc.identifier.citation | Sekar, D., Tusubira, D., & Ross, K. (2022). Corrigendum: TDP-43 and NEAT long non-coding RNA: Roles in neurodegenerative disease. Frontiers in Cellular Neuroscience, 16. | en_US |
dc.identifier.uri | http://ir.must.ac.ug/xmlui/handle/123456789/3086 | |
dc.description.abstract | “Understanding and ameliorating neurodegenerative diseases represents a key challenge for supporting the health span of the aging population. Diverse protein aggregates have been implicated in such neurodegenerative disorders, including amyloid-b, a-synuclein, tau, fused in sarcoma (FUS), and transactivation response element (TAR) DNA-binding protein 43 (TDP-43). Recent years have seen significant growth in our mechanistic knowledge of relationships between these proteins and some of the membrane-less nuclear structures that fulfill key roles in the cell function. These include the nucleolus, nuclear speckles, and paraspeckles | en_US |
dc.language.iso | en_US | en_US |
dc.publisher | Frontiers in Cellular Neuroscience | en_US |
dc.subject | TDP-43 | en_US |
dc.subject | Long non-coding RNA | en_US |
dc.subject | NEAT1 | en_US |
dc.subject | Neurons | en_US |
dc.subject | Paraspeckles | en_US |
dc.subject | TAR DNA-binding protein 43 | en_US |
dc.subject | Nucleic acid therapies | en_US |
dc.subject | Swimming microrobots | en_US |
dc.title | Corrigendum: TDP-43 and NEAT long non-coding RNA: Roles in neurodegenerative disease | en_US |
dc.type | Article | en_US |